Hi Lizzy,
Lizzy wrote:Tony,
I think what I was trying to describe is that these hikers are losing alot of sweat and hence sodium- so by drinking H20 they are replacing fluid lost but not sodium. The body usually maintains a homeostatic balance- but like all balances with the right conditions they can reach the stage where the body cannot adequately compensate. In addition, perhaps the body is actually making itself more hyponatraemic by holding onto water (rather than peeing it out) to protect circulatory volume.
Symptoms of hyponatraemia can also be similar to heat stroke- dizzyness, nause, vomiting, headaches, confusion, muscle cramps- it would be easy to treat incorrectly.
Wow this is getting the brain in gear...
From my understanding over hydration and Hyponatraemia are closely linked, below is an article about Exercise-associated hyponatraemia (EAH) that appeared in New Scientist Jan 9 2010, it was part of a larger article on exercising and was under the heading of MYTH, 'You need to 'push fluids'.
Tony
'You need to 'push fluids'Everyone knows the importance of keeping hydrated. Whether it's a water bottle or a sports drink, athletes and gym bunnies are rarely seen without a source of fluid close at hand. Common advice is to deliberately drink beyond what thirst dictates, or "push fluids", to combat dehydration and keep performance up to scratch.
Usually that's a waste of time, and just occasionally it can be fatal. Exercise-associated hyponatraemia (EAH) is a dangerous condition that occurs when people have drunk so much that the concentration of sodium in their blood falls too low. This leads to excess water moving into the tissues of the brain, causing brain swelling. Symptoms include nausea, vomiting and confusion. In rare cases - 12 have been recorded worldwide - the victim has died.
Slower marathon runners, who tend to drink more over the several hours it takes them to complete the course, are one group more likely to develop EAH , and women seem to be more at risk than men.
The condition can even be caused by sports drinks claimed to be “isotonic” –meaning that they contain they contain the same concentration of dissolved substances as normal body fluids – as they tend to contain sugar but very little salt.
Cases of EAH rose in the US in the 1990s. Tim Noakes, director of exercise science and sports medicine unit at the University of Cape Town, South Africa, who was first to descrbe the condition, blames the rise on marketing activities by the makers of sports drinks, which he says promote overdrinking (British Journal os Sports Medicine, vol 40, p567). He also claims that guidelines issued by the American College of Sports Medicine and other bodies have been influenced by sponsorship from the manufacturers of Gatorade, formerly Quaker Oats, now PepsiCo.
Since awareness of EAH has grown, most guidelines now warn athletes not to overdrink. Although some examples of advice to drink “as much as possible” remain, most advice now sets lower and upper limits on how much to drink. Ron Maughan, a physiologist and sports nutrition specialist at Loughborough University in the UK, says blanket guidelines are flawed because people vary in how much they sweat. He recommends that people weigh themselves before and after their exercise to find out how much they sweat, and drink enough to maintain their body weight.
But this is overdoing it, says Noakes. Drinking to satisfy your thirst is all that is needed. “The easiest way to lower your performance is to overdrink, not underdrink.”